Critical Care Concepts for NCLEX-RN 2024

Critical care concepts are essential for the NCLEX-RN exam as they equip nurses with the knowledge and skills to manage life-threatening conditions. Understanding these concepts ensures safe, effective patient care, improving outcomes in high-stakes environments like ICU and emergency setting.

Endotracheal tube (ETT): Needs Critical Care:

Ventilators: (Critical care needs):

  • The ICU nurse is caring for a sedated client on a ventilator. The ventilator alarm is beeping persistently for low pressure despite the fact the client is calm and has stable vitals. In that case, the most appropriate action for the RN to take first is to check the tubing for holes.
  • If the patient’s presentation and vitals are stable, the nurse should check for any apparent equipment malfunction. If no air leaks or kinks are immediately identifiable, the nurse should call respiratory therapy or the rapid response team (RRT).
  • Persistent alarms despite stable vitals may indicate the patient is trying to talk, or is developing a pneumothorax from increased intrathoracic pressure, or is biting/gagging on the endotracheal tube, or is experiencing bronchospasms. These alarms should never be ignored or turned off, as they may indicate early signs of a change in the patient’s condition.
Pressure in the circuit is too high (HIGH) Ventilator alarmPressure in the circuit is too low (LOW) Ventilator Alarm
CAUSES:
Client coughing
Gagging
Bronchospasm
Fighting the ventilator
ETT occlusion
Kink in the tubing
Increased secretions
Thick secretions
Water in ventilator circuit
CAUSES:
Tubbing is disconnected
Loose connections
Leak
Extubation
Cuffed ETT or trach is deflated
Poorly fitting CPAP/BiPAP mask




  • Intracranial Pressure:
    • The pressure inside of the skull
    • Normal = 5-15
  • Causes of increased ICP:
    • Tumor
    • Bleeding
    • Hydrocephalus
    • Edema
  • Signs and symptoms of Increased ICP:
    • Headache: Often worse in the morning and exacerbated by activities that increase intracranial pressure, such as coughing or straining.
    • Nausea and Vomiting: Especially vomiting that is sudden and without nausea (projectile vomiting).
    • Altered Mental Status: Confusion, lethargy, decreased level of consciousness, and, in severe cases, coma.
    • Papilledema: Swelling of the optic disc, visible upon eye examination.
    • Visual Disturbances: Blurred vision, double vision (diplopia), and transient visual obscurations.
    • Cushing’s Triad: Hypertension, bradycardia, and irregular respirations; a late sign indicating brainstem compression.
    • Seizures: New-onset seizures can be a symptom of increased ICP.
    • Pupil Changes: Unequal pupils (anisocoria) and sluggish or non-reactive pupils, indicating pressure on cranial nerves.

Glasgow Coma Scale

  • Normal = 15
    • This client is fully oriented!
  • Mild injury = 13-15
  • Moderate injury = 9-12
  • Severy injury = <8
  • Less than eight; intubate!
Glasgow Coma Scale Normal= 15
  • Posturing:
    • Decorticate:
      • Abnormal flexion
      • Arms pulled in towards center
      • Clenched fists
      • Rigid muscles
      • Damage to the midbrain
    • Decerebrate:
      • Abnormal extension
      • Arms and legs straight out
      • Toes pointed downwards
      • Neck and had arched back
      • Rigid muscles
      • Damage to deep brain structures: pons
      • Extension of the arms and legs indicates decerebrate posturing, an indication of increased intracranial pressure
Decorticate and Decerebrate

Vasoactive Infusions: (Critical care concept):

Common Indications:
● Cardiac arrest
● Hypotension
● Shock refractory to fluid resuscitation
● Cardiac disease (Acquired or Congenital)

  1. Epinephrine
    • Most often used in cold shock
    • Low doses act on beta-1 receptors
    • Increase the cardiac output
    • High doses act on alpha-1 receptors
    • Increase systemic vascular resistance → increase BP
  2. Norepinephrine
    • Most often used in warm shock
    • Acts on alpha-1 receptors
    • Causes peripheral vasoconstriction → increases BP
    • Increases cardiac output
  3. Dopamine:
    • Used in trauma patients and cold shock
    • Low doses used in kidney failure to increase renal blood flow (‘Renal dopa’)
    • Low doses increase contractility → Increase CO
    • Higher doses cause vasoconstriction → Increase SVR → Increase BP
  4. Phenylephrine:
    • Used for anesthesia-induced hypotension
    • Second line agent in some shock patients
    • Only acts on alpha-1 receptors
    • Causes only vasoconstriction – no inotropy
    • Vasoconstriction → Increased BP
  5. Milrinone:
    • Used in patients with:
      • Cardiogenic shock
      • Decreased cardiac output
      • Congenital/acquired heart defects
    • Causes systemic vasodilation, pulmonary vasodilation, decreased afterload, & increased contractility.
    • Doesn’t increase oxygen consumption
  6. Vasopressin:
    • Antidiuretic hormone (ADH)
    • ANTI-diuresis…. Less diuresis → more volume IN the vascular system.
    • More volume → more pressure!
    • Raises BP
    • Second line in vasodilatory shock
    • Third line in septic shock
      • 1st: Dopa or Norepi
      • 2nd: Epi or phenylephrine
      • 3rd: Vaso

Total Parenteral Nutrition also called hyperalimentation

  • Nutrition delivered intravenously
  • Contains:
    • Dextrose
    • Amino acids
    • Electrolytes
  • Central line is preferred
  • Indications of TPN:
    • Enteral nutrition is contraindicated
    • Client is not tolerating enteral nutrition
    • High risk for aspiration
    • GI tract obstruction
  • Complications:
    • Big infection risk. Scrub that hub! Wash your hands!! Gloves!!!
    • Tubing is changed every day
    • Refrigerated until ready to hang
    • Fluid overload
    • Hyper OR hypoglycemia
    • Do not turn on or off suddenly
    • Titrate
      • Check blood glucose levels
    • Embolism

Parenteral Nutrition is a form of nutrition and is used when there is no other nutritional alternative. Administering nutrition orally or through a nasogastric tube is usually initiated first, before PN is initiated.

  1. A resuscitation (Ambu) bag needs to be kept at the bedside of a client with an endotracheal tube or a tracheostomy tube at all times.
  2. Never insert a plug (cap) into a tracheostomy tube until the cuff is deflated and the inner cannula is removed; prior insertion prevents airflow to the client.
  3. If the chest tube is pulled out of the chest accidently, pinch the skin opening together, apply an occlusive sterile dressing, cover the dressing with overlapping pieces of 2-inch (5cm) tape, and call the PHCP immediately.
  4. The airway is the priority concern in an inhalation injury.
  5. A client who is hypoxemic and has chronic hypercapnia may require low levels of oxygen delivery at 1 to 2L /minute. because a low arterial oxygen level is the client’s primary drive for breathing; always check PHCP’s prescriptions.
  6. A bag-valve resuscitation mask should be available at the bedside for all clients receiving mechanical ventilation.
  7. For all clients receiving mechanical ventilation, always assess the client first and then assess the ventilator.
  8. Never set the ventilator alarm controls to the off position.
  9. Clients with a respiratory disorder should be positioned with the head of the bed elevated.
  10. Acute cardiac tamponade can occur when small volumes (20-50mL) of fluid accumulate rapidly in the pericardium.
  11. For the client with Increased ICP, elevate the head of the bed 30-40 degrees, avoid Trendelenburg’s position, and prevent flexion of the neck and hips.
  12. Abrupt discontinuation of a PN solution can result in hypoglycemia. The flow rate should be decreased gradually when the PN is discontinued.

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